To decide whether or not the absence of RGS2 alters tubular sodium reabsorption, wild kind and RGS2-/- mice were subjected to acute plasma quantity growth by ongoing infusion of isotonic saline
To stimulate the excretion of extra h2o and sodium, arterial blood stress was increased by systemic administration of the non-selective nitric oxide synthase inhibitor, L-Name right after baseline recordings. As…